However, there are indications that the risk of thrombosis in patients being treated at home is significantly lower – although not quite zero. ![]() ![]() We first have to wait for ongoing studies to be completed. There are still no guidelines when it comes to prophylactic anticoagulant therapy for COVID-19 out-patients. What do these new findings mean for patients dealing with the disease at home? However, as of yet, there is no conclusive evidence in favor of this. One course of action currently being discussed for seriously ill patients is increasing the dosage of thrombosis prophylaxis up to a therapeutic dose. Scientific organizations generally recommend anticoagulants for COVID-19 patients being treated in hospital, particularly those in intensive care – and, of course, for patients who have experienced thrombosis before. These drugs can of course also be used with bedridden coronavirus patients, preferably with the anticoagulant heparin. Thrombosis prophylaxis is usually recommended for internal illnesses that render patients bedridden – but this is a general rule and not specific to COVID-19. ![]() However, we are still waiting on the results of randomized, controlled clinical trials, which are still underway.Īre there any official recommendations on how to administer anticoagulants to patients with COVID-19? We now have the results of an observational study of over 2,700 COVID-19 patients in the United States, which suggest that anticoagulant therapy significantly reduces hospital mortality in coronavirus patients. We’re only in the early stages of this scientific investigation.Ĭan blood thinning medications reduce these risks? Apart from that, the exact origin is still largely unclear. Specific risk factors are likely to be already-damaged blood vessels, e.g. Of course, there are also a number of well-known risk factors – such as old age, obesity and diabetes mellitus. Generally speaking, we can say it is mainly seriously ill COVID-19 patients who are affected – in other words, patients whose bodies have already sustained a great deal of damage. This can lead to organ failure caused by the clots blocking the vessels.Īre there factors that stimulate thrombosis in coronavirus patients? This goes hand in hand with a severe inflammatory reaction, causing clots to form in the patients’ large and, in some cases, small blood vessels – and sometimes even in more than one place. However, generally speaking, we can now say that COVID-19 seriously damages the inner walls of the blood vessels, known as the endothelial cells. What exactly does the coronavirus do to the body? ![]() Most viral diseases have no effect on thrombosis. There are viruses that do just the opposite, causing patients to hemorrhage. This is much higher than the usual rate of cases in patients with bacterial pneumonia, for example.Īre viral diseases known to be a potential cause of thrombosis? A Dutch publication analyzing data from 184 patients found thrombosis in a third of people infected with COVID-19. More evidence has come to light since then, however. Initially, no one suspected there was a link, as everyone assumed the coronavirus was a pulmonary virus that attacks the lungs and causes serious respiratory infections. What is your perspective on these new findings?ĭuring the acute infection phase – particularly the first few weeks – doctors and professors who we’ve collaborated with for many years would call me every day, reporting cases of thrombosis or pulmonary embolisms in their COVID-19 patients. Misselwitz, there are indications that patients with COVID-19 are susceptible to blood clots, which can lead to embolisms or, in layman’s terms, the obstruction of the blood vessels.
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